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samples was lower when testing against E. coli than when testing against S. aureus. This result is similar to that found by Kim and others (2007). These results can be explained on the basis of the differences in the cell wall composition of each strain explained as section 2.2.1.1 Mechanisms of NPs against bacteria. Another study showed similar results between E. coli, S. aureus and Ag NPs (Liu 2010). However, Liu’s study also shows that smaller Ag NPs had better antibacterial effects but higher cytotoxicity. In 2011, a study done by Guzman and others showed antibacterial activity of Ag NPs against not only E. coli and S. aureus, but also, Pseudomonas aeruginosa. The average particle sizes used were 9 ± 2, 14 ± 5, 24 ± 6, and 30 ± 7 nm and the antibacterial activity was measured by the Kirby-Bauer method. As a result, the presence of NPs at a certain level inhibited bacterial growth by more than 90%, and clear- zone diameters increased as the concentration of Ag NPs increased in all three bacterial strains. To determine the MIC of Ag NPs against bacterial strains, a standard dilution micromethod was also performed. The smaller Ag NPs, 9 ± 2 and 14 ± 5 nm, showed considerable antibacterial activity. The MIC of Ag NPs on P. aeruginosa and E. coli were lower than that of S. aureus. More studies have been reported concerning the antimicrobial effects of Ag NPs on a broad spectrum of microorganisms. It is well recognized that the higher the concentration and the smaller the particle size the greater the inhibitory effect on microorganisms. However, the mechanisms underlying inhibitory effects are not yet completely understood. 12PDF Image | ZINC OXIDE AND SILVER NANOPARTICLES ON INTESTINAL BACTERIA
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